940 research outputs found
Loss of heterozygosity in multistage carcinogenesis of esophageal carcinoma at high-incidence area in Henan Province, China
- Publication venue
- 'Baishideng Publishing Group Inc.'
- Publication date
- 01/01/2005
- Field of study
Aim: Microsatellites are the repeated DNA sequences scattered widely within the genomes and closely linked with many important genes. This study was designed to characterize the changes of microsatellite DNA loss of heterozygosity (LOH) in esophageal carcinogenesis. Methods: Allelic deletions in 32 cases of matched precancerous, cancerous and normal tissues were examined by syringe microdissection under an anatomic microscope and microsatellite polymorphism analysis using 15 polymorphic markers on chromosomes 3p, 5q, 6p, 9p, 13q, 17p, 17q and 18q. Results: Microsatellite DNA LOH was observed in precancerous and cancerous tissues, except D9S1752. The rate of LOH increased remarkably with the lesions progressed from basal cell hyperplasia (BCH) to squamous cell carcinoma (SCC) (P60%). LOH loci were different in precancerous and cancerous tissues. LOH in D3S1234 and TP53 was the common event in different lesions from the same patients. Conclusion: Microsatellite DNA LOH occurs in early stage of human esophageal carcinogenesis, even in BCH. With the lesion progressed, gene instability increases, the accumulation of this change may be one of the important mechanisms driving precancerous lesions to cancer. © 2005 The WJG Press and Elsevier Inc. All rights reserved.published_or_final_versio
STUDY OF PALLADIUM ANALYTE BINARY-SYSTEM IN THE GRAPHITE-FURNACE BY SURFACE ANALYTICAL TECHNIQUES
- Publication venue
- Publication date
- 01/04/1992
- Field of study
The mechanisms of the stabilization of Pb, Zn and As by a palladium modifier were investigated by scanning electron microscopy, X-ray diffraction spectrometry and X-ray photoelectron spectrometry. All three elements were found to form an intermetallic solid solution with excess of Pd. The concentration ratio of Pd to analyte varies with the surface depth of Pd-analyte species. Chemical shifts measured in the binding energy of Pd and analyte are insignificant. The monophase of the Pd lattice in which some Pd atoms are displaced by the analyte atoms has been identified as being dominant. It is suggested that the reduced Pd and analyte form a stable intermetallic solid solution during the ashing stage and the analyte atoms remain in the Pd lattice until the temperature is high enough to break down the lattice
食管癌原發灶與淋巴結轉移灶細胞染色體變化特征的比較
- Publication venue
- China (中國)
- Publication date
- 01/01/2005
- Field of study
BACKGROUND & OBJECTIVE: Local lymph node and blood metastasis could occur at early stage of esophageal squamous cell carcinoma (ESCC), which may be the key factors of its recurrence and poor prognosis. However, the mechanism of ESCC metastasis is unclear. This study was to analyze the genetic changes in primary lesion and lymph node metastases of ESCC, to screen for and locate ESCC metastasis-related genes. METHODS: Genomic alterations in 15 pairs of primary lesions and matched metastatic lymph nodes of ESCC were analyzed by comparative genomic hybridization (CGH). RESULTS: In the 15 pairs of tissues, the most common chromosomal alterations were the gains of 3q, 8q, 6p, 20p, 5p, 18p, 2p, 2q and 1q, and the losses of 10p, 10q, 17p, 18q, 4p and 13q. Of these changes, the most significant finding was the gain of 6p with a frequency of 47% in metastatic lymph nodes and 13% in primary lesions, and the gain of 20p with a frequency of 73% in metastatic lymph nodes and 33% in primary lesions. The second interesting finding was the loss of 10p with a frequency of 53% in metastatic lymph nodes and 13% in primary lesions, and the loss of 10q with a frequency of 47% in metastatic lymph nodes and 13% in primary lesions. CONCLUSION: The gains of 6p and 20p and the losses of 10p and 10q are common genomic alterations in primary lesion and lymph node metastases of ESCC, which may code ESCC metastasis-related genes.背景與目的:食管癌早期可發生局部淋巴或血行轉移,這是導致復發和預后差的主要原因。但是,食管癌轉移發生的分子機制尚不清楚。本研究旨在分析食管癌原發灶和淋巴結轉移灶腫瘤細胞染色體變化的特征,尋找或定位與食管癌轉移相關基因,加深對其轉移機制的了解。方法:應用比較基因組雜交技術(comparativegenomichybridization,CGH)分析15例食管癌患者原發灶和其對應的淋巴結轉移灶的染色體基因組改變。結果:最常見染色體DNA拷貝數增加的部位是3q,8q,6p,20p,5p,18p,2p,2q,1q;常見的染色體DNA拷貝數丟失的部位是10p,10q,17p,18q,4p,13q。其中,最有意義的發現是6p增加(原發灶:2/15,13%,轉移灶:7/15,47%),20p增加(原發灶:5/15,33.3%,轉移灶:11/15,73.3%)。第二個發現是10p丟失(原發灶:2/15,13.3%,轉移灶:8/15,53%),10q丟失(原發灶:2/15,13.3%,轉移灶:7/15,46.6%)。結論:食管癌原發灶和淋巴結轉移灶細胞染色體基因組改變最顯著的部位是6p,20p的增加和10p,10q的丟失;這些部位可能存在與食管癌細胞淋巴結轉移相關的基因。link_to_subscribed_fulltex
Vemurafenib-resistant BRAF selects alternative branch points different from its wild-type BRAF in intron 8 for RNA splicing
- Author
- A Corrionero
- A Corvelo
- A Crisci
- A Kramer
- AJ Taggart
- BR Graveley
- C Montagut
- C Sun
- CM Johannessen
- DA Sterner
- EC Ibrahim
- F Rossi
- FO Desmet
- GA McArthur
- H Davies
- H Malca
- H Sun
- J Hamm
- J Villanueva
- J Vogel
- JA Berglund
- JA Curtin
- JC Noble
- K Gao
- KT Flaherty
- L Cartegni
- M Ajiro
- M Ajiro
- M Salton
- MR Green
- N Kfir
- O Gozani
- PA Mercado
- PB Chapman
- PI Poulikakos
- Q Tran
- R Brosi
- R Reed
- RM Lardelli
- SM Berget
- TR Mercer
- V Majerciak
- XD Fu
- Y Dou
- Y Kondo
- YA Zhuang
- ZM Zheng
- ZM Zheng
- ZM Zheng
- ZM Zheng
- ZM Zheng
- ZM Zheng
- ZM Zheng
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- Field of study
Search for the standard model Higgs boson at LEP
- Author
- Abbaneo D
- Abbaneo D
- Abbiendi G
- Abbiendi G
- Abdallah J
- Abreu P
- Achard P
- Adam W
- Adriani O
- Adzic P
- Affholderbach K
- Aguilar-Benitez M
- Ainsley C
- Akesson PF
- Albrecht T
- Alcaraz J
- Alderweireld T
- Alemanni G
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- Alexander G
- Allaby J
- Allison J
- Allmendinger T
- Allport PP
- Aloisio A
- Alviggi MG
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- Amaral P
- Amato S
- Anagnostou G
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- Anderhub H
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- Anselmo F
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- Apel WD
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- Arefiev A
- Armstrong SR
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- Ask S
- Asman B
- Augustin JE
- Augustinus A
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- Axen D
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- Azzurri P
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- Bailey I
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- Yang BZ
- Yang CG
- Yang HJ
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- Zachariadou K
- Zalewska A
- Zalewski P
- Zalite A
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- Zavrtanik D
- Zeitnitz C
- Zer-Zion D
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- Zhu GY
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- Zhuang HL
- Zhuravlov V
- Zichichi A
- Ziegler T
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- Zintchenko A
- Zito G
- Zivkovic L
- Zobernig G
- Zoller M
- Zupan M
- Publication venue
- ELSEVIER SCIENCE BV
- Publication date
- 17/07/2003
- Field of study
Search for new phenomena in final states with an energetic jet and large missing transverse momentum in pp collisions at √ s = 8 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abbott DC
- Abeling K
- Abhayasinghe DK
- Abidi SH
- AbouZeid OS
- Abraham NL
- Abramowicz H
- Abreu H
- Abud AA
- Abulaiti Y
- Acharya BS
- Achkar B
- Adachi S
- Adam L
- Adamczyk L
- Adamek L
- Adelman J
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- Akesson TPA
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- Alberghi GL
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- Aleksandrov IN
- Alexa C
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- Ali B
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- Alimonti G
- Allaire C
- Allbrooke BMM
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- Alviggi MG
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- de Renstrom PAB
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- Denisov SP
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- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2015
- Field of study
Results of a search for new phenomena in final states with an energetic jet and large missing transverse momentum are reported. The search uses 20.3 fb−1 of √ s = 8 TeV data collected in 2012 with the ATLAS detector at the LHC. Events are required to have at least one jet with pT > 120 GeV and no leptons. Nine signal regions are considered with increasing missing transverse momentum requirements between Emiss T > 150 GeV and Emiss T > 700 GeV. Good agreement is observed between the number of events in data and Standard Model expectations. The results are translated into exclusion limits on models with either large extra spatial dimensions, pair production of weakly interacting dark matter candidates, or production of very light gravitinos in a gauge-mediated supersymmetric model. In addition, limits on the production of an invisibly decaying Higgs-like boson leading to similar topologies in the final state are presente
Search for direct pair production of the top squark in all-hadronic final states in proton-proton collisions at s√=8 TeV with the ATLAS detector
- Author
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- Abdallah J
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- Wosiek BK
- Wotschack J
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- Yusuff I
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- Zaidan R
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- Zanzi D
- Zeitnitz C
- Zeman M
- Zemla A
- Zengel K
- Zenin O
- Zenis T
- Zerwas D
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- Zhang Z
- Zhao Z
- Zhemchugov A
- Zhong J
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- Zhu CG
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- Zhukov K
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zurzolo G
- Zutshi V
- Zwalinski L
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2014
- Field of study
The results of a search for direct pair production of the scalar partner to the top quark using an integrated luminosity of 20.1fb−1 of proton–proton collision data at √s = 8 TeV recorded with the ATLAS detector at the LHC are reported. The top squark is assumed to decay via t˜→tχ˜01 or t˜→ bχ˜±1 →bW(∗)χ˜01 , where χ˜01 (χ˜±1 ) denotes the lightest neutralino (chargino) in supersymmetric models. The search targets a fully-hadronic final state in events with four or more jets and large missing transverse momentum. No significant excess over the Standard Model background prediction is observed, and exclusion limits are reported in terms of the top squark and neutralino masses and as a function of the branching fraction of t˜ → tχ˜01 . For a branching fraction of 100%, top squark masses in the range 270–645 GeV are excluded for χ˜01 masses below 30 GeV. For a branching fraction of 50% to either t˜ → tχ˜01 or t˜ → bχ˜±1 , and assuming the χ˜±1 mass to be twice the χ˜01 mass, top squark masses in the range 250–550 GeV are excluded for χ˜01 masses below 60 GeV
Search for pair-produced long-lived neutral particles decaying to jets in the ATLAS hadronic calorimeter in ppcollisions at √s=8TeV
- Author
- Aad G
- Abbott B
- Abdallah J
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- Sundermann JE
- Suruliz K
- Susinno G
- Sutton MR
- Suzuki Y
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- Swedish S
- Swiatlowski M
- Sykora I
- Sykora T
- Ta D
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- Taenzer J
- Taffard A
- Tafirout R
- Taiblum N
- Takai H
- Takashima R
- Takeda H
- Takeshita T
- Takubo Y
- Talby M
- Talyshev AA
- Tam JYC
- Tan KG
- Tanaka J
- Tanaka R
- Tanaka S
- Tanaka S
- Tanasijczuk AJ
- Tannenwald BB
- Tannoury N
- Tapprogge S
- Tarem S
- Tarrade F
- Tartarelli GF
- Tas P
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- Tashiro T
- Tassi E
- Tayalati Y
- Taylor FE
- Taylor GN
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- Tehrani FS
- Teischinger FA
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- Temming KK
- Ten Kate H
- Teng PK
- Teoh JJ
- Terada S
- Terashi K
- Terron J
- Terzo S
- Testa M
- Teuscher RJ
- Therhaag J
- Theveneaux-Pelzer T
- Thomas JP
- Thomas-Wilsker J
- Thompson AS
- Thompson EN
- Thompson PD
- Thompson PD
- Thompson RJ
- Thomsen LA
- Thomson E
- Thomson M
- Thong WM
- Thun RP
- Tian F
- Tibbetts MJ
- Tikhomirov VO
- Tikhonov YA
- Timoshenko S
- Tiouchichine E
- Tipton P
- Tisserant S
- Todorov T
- Todorova-Nova S
- Toggerson B
- Tojo J
- Tokar S
- Tokushuku K
- Tollefson K
- Tolley E
- Tomlinson L
- Tomoto M
- Tompkins L
- Toms K
- Topilin ND
- Toro RC
- Torregrosa EF
- Torrence E
- Torres H
- Torro Pastor E
- Toth J
- Touchard F
- Tovey DR
- Tran HL
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- Tremblet L
- Tricoli A
- Trigger IM
- Trincaz-Duvoid S
- Tripiana MF
- Trischuk W
- Trocme B
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- Trottier-McDonald M
- Trovatelli M
- True P
- Trzebinski M
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- Tsarouchas C
- Tseng JC-L
- Tsiareshka PV
- Tsionou D
- Tsipolitis G
- Tsirintanis N
- Tsiskaridze S
- Tsiskaridze V
- Tskhadadze EG
- Tsukerman I
- Tsulaia V
- Tsuno S
- Tsybychev D
- Tudorache A
- Tudorache V
- Tuna AN
- Tupputi SA
- Turchikhin S
- Turecek D
- Turra R
- Tuts PM
- Tykhonov A
- Tylmad M
- Tyndel M
- Uchida K
- Ueda I
- Ueno R
- Ughetto M
- Ugland M
- Uhlenbrock M
- Ukegawa F
- Unal G
- Undrus A
- Unel G
- Ungaro FC
- Unno Y
- Unverdorben C
- Urbaniec D
- Urquijo P
- Usai G
- Usanova A
- Vacavant L
- Vacek V
- Vachon B
- Valencic N
- Valentinetti S
- Valero A
- Valery L
- Valkar S
- Vallecorsa S
- Van Den Wollenberg W
- Van Der Deijl PC
- van der Geer R
- van der Graaf H
- Van Der Leeuw R
- van der Ster D
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- Van Gemmeren P
- van Huysduynen LH
- Van Nieuwkoop J
- van Vulpen I
- Van Woerden MC
- Vanadia M
- Vandelli W
- Vanguri R
- Vaniachine A
- Vankov P
- Vannucci F
- Vaque FV
- Vardanyan G
- Vari R
- Varnes EW
- Varol T
- Varouchas D
- Vartapetian A
- Varvell KE
- Vazeille F
- Vazquez JGP
- Veatch J
- Veloso F
- Velz T
- Veneziano S
- Ventura A
- Ventura D
- Venturi M
- Venturi N
- Venturini A
- Vercesi V
- Verducci M
- Verkerke W
- Vermeulen JC
- Verzini MJA
- Vest A
- Vetterli MC
- Viazlo O
- Vichou I
- Vickey T
- Viehhauser GHA
- Viel S
- Vigne R
- Villa M
- Vilucchi E
- Vincter MG
- Vinogradov VB
- Virzi J
- Vivarelli I
- Vlachos S
- Vladoiu D
- Vlasak M
- Vogel A
- Vokac P
- Volpi G
- Volpi M
- von der Schmitt H
- von Radziewski H
- von Toerne E
- Vorobel V
- Vorobev K
- Vos M
- Voss R
- Vossebeld JH
- Vranjes N
- Vrba V
- Vreeswijk M
- Vuanh T
- Vuillermet R
- Vukotic I
- Vykydal Z
- Wagner P
- Wagner W
- Wahlberg H
- Wahrmund S
- Wakabayashi J
- Walder J
- Walker R
- Walkowiak W
- Wall R
- Waller P
- Walls FMG
- Walsh B
- Wang C
- Wang C
- Wang F
- Wang H
- Wang H
- Wang J
- Wang J
- Wang K
- Wang R
- Wang SM
- Wang T
- Wang X
- Wanotayaroj C
- Warburton A
- Ward CP
- Wardrope DR
- Warsinsky M
- Washbrook A
- Wasicki C
- Watkins PM
- Watson AT
- Watson IJ
- Watson MF
- Watts G
- Watts S
- Waugh BM
- Webb S
- Weber MS
- Weber SW
- Webster JS
- Weidberg AR
- Weigell P
- Weinert B
- Weingarten J
- Weiser C
- Weits H
- Wells PS
- Wemans ADV
- Wenaus T
- Wendland D
- Weng Z
- Wengler T
- Wenig S
- Wermes N
- Werner M
- Werner P
- Wessels M
- Wetter J
- Whalen K
- White A
- White MJ
- White R
- White S
- Whiteson D
- Wicke D
- Wickens FJ
- Wiedenmann W
- Wielers M
- Wienemann P
- Wiglesworth C
- Wiik-Fuchs LAM
- Wijeratne PA
- Wildauer A
- Wildt MA
- Wilkens HG
- Will JZ
- Williams HH
- Williams S
- Willis C
- Willocq S
- Wilson A
- Wilson JA
- Wingerter-Seez I
- Winklmeier F
- Winter BT
- Wittgen M
- Wittig T
- Wittkowski J
- Wollstadt SJ
- Wolter MW
- Wolters H
- Wong KHY
- Wosiek BK
- Wotschack J
- Woudstra MJ
- Wozniak KW
- Wright M
- Wu M
- Wu SL
- Wu X
- Wu Y
- Wulf E
- Wyatt TR
- Wynne BM
- Xella S
- Xiao M
- Xu D
- Xu L
- Yabsley B
- Yacoob S
- Yakabe R
- Yamada M
- Yamaguchi H
- Yamaguchi Y
- Yamamoto A
- Yamamoto K
- Yamamoto S
- Yamamura T
- Yamanaka T
- Yamauchi K
- Yamazaki Y
- Yan Z
- Yang H
- Yang H
- Yang UK
- Yang Y
- Yanush S
- Yao L
- Yao W-M
- Yasu Y
- Yatsenko E
- Ye J
- Ye S
- Yeletskikh I
- Yen AL
- Yildirim E
- Yildiz HD
- Yilmaz M
- Yoosoofmiya R
- Yorita K
- Yoshida R
- Yoshihara K
- Young C
- Young CJS
- Youssef S
- Yu DR
- Yu J
- Yu J
- Yu JM
- Yuan L
- Yurkewicz A
- Yusuff I
- Zabinski B
- Zaidan R
- Zaitsev AM
- Zaman A
- Zambito S
- Zanello L
- Zanzi D
- Zeitnitz C
- Zeman M
- Zemla A
- Zengel K
- Zenin O
- Zenis T
- Zerwas D
- Zhang D
- Zhang F
- Zhang H
- Zhang J
- Zhang L
- Zhang X
- Zhang Z
- Zhao Z
- Zhemchugov A
- Zhong J
- Zhou B
- Zhou L
- Zhou N
- Zhu CG
- Zhu H
- Zhu J
- Zhu Y
- Zhuang X
- Zhukov K
- Zibell A
- Zieminska D
- Zimine NI
- Zimmermann C
- Zimmermann R
- Zimmermann S
- Zimmermann S
- Zinonos Z
- Ziolkowski M
- Zobernig G
- Zoccoli A
- zur Nedden M
- Zurzolo G
- Zutshi V
- Zwalinski L
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2014
- Field of study
The ATLAS detector at the Large Hadron Collider at CERN is used to search for the decay of a scalar boson to a pair of long-lived particles, neutral under the Standard Model gauge group, in 20.3fb−1of data collected in proton–proton collisions at √s=8TeV. This search is sensitive to long-lived particles that decay to Standard Model particles producing jets at the outer edge of the ATLAS electromagnetic calorimeter or inside the hadronic calorimeter. No significant excess of events is observed. Limits are reported on the product of the scalar boson production cross section times branching ratio into long-lived neutral particles as a function of the proper lifetime of the particles. Limits are reported for boson masses from 100 GeVto 900 GeV, and a long-lived neutral particle mass from 10 GeVto 150 GeV
An overview of cancer/testis antigens expression in classical Hodgkin's lymphoma (cHL) identifies MAGE-A family and MAGE-C1 as the most frequently expressed antigens in a set of Brazilian cHL patients
- Author
- A Diepstra
- AA Jungbluth
- AA Jungbluth
- Achim A Jungbluth
- Adriana B Bortoluzzo
- AH Campos
- Antonio C Alves
- B Yang
- BF Skinnider
- CV Hedvat
- D Atanackovic
- D Atanackovic
- D Perez
- Denise Frosina
- E Yakirevich
- EF Velazquez
- EM Souza
- F Bellati
- Gisele WB Colleoni
- GW Colleoni
- H Chambost
- HA Vaughan
- HJ Heidebrecht
- J Kim
- J Kononen
- J Vassallo
- Jodie Tassello
- JS Goydos
- L Brepoels
- LG Almeida
- M Monte
- Mariane CG Assis
- MO Riener
- MV Dhodapkar
- Nicole C Hanson
- OL Caballero
- Otávio CG Baiocchi
- P Sharma
- PT Nelson
- R Zhuang
- Riguel J Inaoka
- S Bai
- S Poppema
- SH Swerdlow
- SM Oba-Shinjo
- T Nagao
- T Peikert
- T Suyama
- VC Andrade
- X Zhu
- YT Chen
- YT Chen
- ZM Cilensek
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
<p>Abstract</p> <p>Background</p> <p>Cancer/testis antigens are considered potential targets for immunotherapy due to their tumor-associated expression pattern. Although recent studies have demonstrated high expression of CT45 in classical Hodgkin's lymphomas (cHL), less is known about the expression pattern of other families of CTAs in cHL. We aim to evaluate the expression of MAGE-A family, MAGE-C1/CT7, MAGE-C2/CT10, NY-ESO1 and GAGE family in cHL and to correlate their expression with clinical and prognostic factors in cHL.</p> <p>Methods</p> <p>Tissue microarray was generated from 38 cHL archival cases from Pathology Department of Universidade Federal de Sao Paulo. Immunohistochemistry (IHC) was done using the following panel of antibodies: MAGE-A family (MA454, M3H67, 57B and 6C1), GAGE (#26), NY-ESO-1 (E978), MAGE-C1/CT7 (CT7-33) and MAGE-C2/CT10 (CT10#5).</p> <p>Results</p> <p>We found CTA expression in 21.1% of our cHL series. Among the tested CTAs, only MAGE-A family 7/38 (18.4%) and MAGE-C1/CT7 5/38 (13.2%) were positive in our cHL samples. We found higher CTA positivity in advanced stage (28.6%) compared to early stage (11.8%) disease, but this difference was not statistically significant. Analysis of other clinicopathological subgroups of cHL including histological subtypes, EBV status and response to treatment also did not demonstrate statistical significant differences in CTA expression.</p> <p>Conclusion</p> <p>We found CTA expression in 21.1% of cHL samples using our panel. Our preliminary findings suggest that from all CTAs included in this study, MAGE-A family and MAGE-C1/CT7 are the most interesting ones to be explored in further studies.</p
Genetic variation and evolutionary demography of Fenneropenaeus chinensis populations, as revealed by the analysis of mitochondrial control region sequences
- Author
- Avise JC
- Avise JC
- Bandelt HJ
- Bandelt HJ
- Cha HK
- Chu KH
- Cui ZX
- Deng JY
- Deng JY
- Deng JY
- Excoffier L
- Excoffier L
- Fu YX
- Grant WS
- Harpending HC
- Hartl DL
- Hasegawa M
- Hedrick PW
- Hey J
- Hey J
- Hualkasin W
- Huelsenbeck JP
- Hwang GL
- Hwang GL
- Jie Kong
- Klinbunga S
- Lee WJ
- Liu JY
- Liu JY
- Liu P
- Liu P
- Liu P
- McMillan-Jackson A
- McMillan-Jackson AL
- Meng XH
- Munro JL
- Nei M
- Posada D
- Quan JX
- Ray N
- Rogers AR
- Rogers AR
- Ronquist F
- Salgueiro P
- Schneider S
- Schneider S
- Shi T
- Shi T
- Slatkin M
- Swofford DL
- Tajima F
- Tamura K
- Thompson JD
- Tsoi KH
- Tzeng TD
- Valles-Jimenez R
- Wang P
- Wang WJ
- Watterson GA
- Wei Shi
- Xiao Yu Kong
- Yu Long Li
- Zhu S
- Zhuang ZM
- Publication venue
- Sociedade Brasileira de Genética
- Publication date
- 01/01/2010
- Field of study
Genetic variation and evolutionary demography of the shrimp Fenneropenaeus chinensis were investigated using sequence data of the complete mitochondrial control region (CR). Fragments of 993 bp of the CR were sequenced for 93 individuals from five localities over most of the species' range in the Yellow Sea and the Bohai Sea. There were 84 variable sites defining 68 haplotypes. Haplotype diversity levels were very high (0.95 ± 0.03-0.99 ± 0.02) in F. chinensis populations, whereas those of nucleotide diversity were moderate to low (0.66 ± 0.36%-0.84 ± 0.46%). Analysis of molecular variance and conventional population statistics (FST ) revealed no significant genetic structure throughout the range of F. chinensis. Mismatch distribution, estimates of population parameters and neutrality tests revealed that the significant fluctuations and shallow coalescence of mtDNA genealogies observed were coincident with estimated demographic parameters and neutrality tests, in implying important past-population size fluctuations or range expansion. Isolation with Migration (IM) coalescence results suggest that F. chinensis, distributed along the coasts of northern China and the Korean Peninsula (about 1000 km apart), diverged recently, the estimated time-split being 12,800 (7,400-18,600) years ago
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